Kenneth J Renner: Influence Statistics

Kenneth J Renner

Kenneth J Renner

Department of Biology, University of South Dakota, Vermillion, SD 57069 USA | Neuroscience Group, Division of Basic Biomedical Sciences, Sanford School of Medicine, University ...

Kenneth J Renner: Expert Impact

Concepts for which Kenneth J Renner has direct influence: Amphetamine withdrawal , Serotonergic activity , Ventral hippocampus , Central nucleus , Serotonin release , Opponent recognition , Monoamine levels .

Kenneth J Renner: KOL impact

Concepts related to the work of other authors for which for which Kenneth J Renner has influence: Social defeat , Male rats , Nucleus accumbens , Sexual behavior , Physical activity , Rainbow trout , 5 ht .

KOL Resume for Kenneth J Renner

Year
2022

Department of Biology, University of South Dakota, Vermillion, SD 57069 USA

2021

Department of Biology, University of South Dakota, Vermillion, SD 57069, USA.

2020

Department of Biology, University of South Dakota, Vermillion, SD 57069, USA

2019

Department of Biology and Neuroscience Group, University of South Dakota, Vermillion, SD 57069, USA. Electronic address:

2018

Center for Brain and Behavior Research, Basic Biomedical Sciences, Sanford School of Medicine, University of South Dakota, Vermillion, South Dakota

2017

Department of Biology, University of South Dakota, Vermillion, SD, USA;, View further author information

2016

Center for Brain and Behavior Research, Department of Biology, University of South Dakota, Vermillion, SD, USA

Department of Biology, University of South Dakota, Vermillion, South Dakota, United States of America

2015

Center for Brain and Behavior Research, University of South Dakota, Vermillion, SD, United States

2014

University of South Dakota Department of Biology & Center for Brain and Behavior Research Vermillion SD USA

2013

Department of Biology, University of South Dakota, Vermillion, SD, USA

2012

University of South Dakota Neuroscience Group Basic Biomedical Sciences Sanford School of Medicine Vermillion SD USA

2011

Department of Biology, University of South Dakota, 57069, Vermillion, SD, USA

2010

Biology Dept, University of South Dakota, SD, USA

Neuroscience Group, Division of Basic Biomedical Sciences, Sanford School of Medicine, University of South Dakota, Vermillion, South Dakota; and

2009

Department of Biology, University of South Dakota

2008

Department of Biology & Neuroscience Group, University of South Dakota, 414 E. Clark St. Vermillion, SD, 57069, USA

2007

Neuroscience Group, Division of Basic Biomedical Sciences, Sanford School of Medicine, University of South Dakota, 414 East Clark Street, Vermillion, SD, USA

Department of Biology, The University of South Dakota, Vermillion, South Dakota 57069

2006

Division of Basic Biomedical Sciences, Neuroscience Group, Sanford School of Medicine at the University of South Dakota, 414 East Clark Street, Vermillion, SD 57069, USA

Department of Biology and Neuroscience, University of South Dakota

2005

Department of Biology and Neuroscience Group, Division of Basic Biomedical Sciences, School of Medicine, The University of South Dakota, Vermillion, SD 57069, USA

2004

Biology Department and Neuroscience Group, University of South Dakota, Vermillion, SD 57069, USA

2003

Biology and Neuroscience, University of South Dakota, Vermillion, SD, USA

2002

Department of Biology and Neuroscience Group, University of South Dakota, 414 East Clark Street, Vermillion, SD 57069-3290, USA

2001

Department of Biology and Neuroscience Group, University of South Dakota, Vermillion, South Dakota, 57069-2390

2000

Department of Biology and Neuroscience Group, University of South Dakota, Vermillion, South Dakota, 57069

1999

Department of Psychology, Hunter College of CUNY, New York, N.Y. and, Department of Biology, University of South Dakota, Vermillion, S. Dak., USA

1997

Department of Biology, The University of South Dakota, Vermillion, SDUSA

1996

Department of Zoology, Oregon State University, Corvallis, Oreg., Biomedical Sciences Department, Southwest Missouri State University Springfield, Mo., USA

1993

Department of Biomedical Sciences, Southwest Missouri State University, Springfield, MO 65804 USA

1992

Department of Biomedical Sciences, Southwest Missouri State University, Springfield, MO 65804, USA

1989

Laboratory of Neuroendocrinology, The Rockefeller University, New York, NY 10021, U.S.A.

1988

Department of Chemistry, University of Kansas, Lawrence, KA 66045 U.S.A.

1987

Department of Chemistry, University of Kansas, Lawrence, KA 66045, U.S.A.

Laboratory of Neuroendocrinology, Rockefeller University, New York, NY 10021 USA

1986

Laboratory of Neuroendocrinology, Rockefeller University, New York, NY 1002 USA

The Rockefeller University, New York, NY 10021, U.S.A.

1985

Rockefeller University, New York, NY 10021 U.S.A.

1984

Department of Physiology and Cell Biology, University of Kansas, Lawrence, KSUSA

Rockefeller University, New York, N.Y. 10021, USA

1981

Department of Physiology and Cell Biology, University of Kansas, Lawrence, KS 66045, USA

Prominent publications by Kenneth J Renner

KOL-Index: 14223 . Decreases in serotonergic activity in the central nucleus of the amygdala reduce responses to stressors, suggesting an important role for serotonin in this region of the amygdala in stress reactivity. However, it is not known whether exposure to stressors actually increases serotonin release in the central nucleus of the amygdala. The current experiment tested the hypothesis that restraint ...
Known for Central Nucleus | Restraint Stress | Serotonin Release | Releasing Factor
KOL-Index: 13075 . Interactions between central corticotropin-releasing factor (CRF) and serotonergic systems are believed to be important for mediating fear and anxiety behaviors. Recently we demonstrated that infusions of CRF into the rat dorsal raphe nucleus result in a delayed increase in serotonin release within the medial prefrontal cortex that coincided with a reduction in fear behavior. The current ...
Known for Dorsal Raphe | Type 2 | Spraguedawley Receptors | Central Nucleus
KOL-Index: 12566 . Acute amphetamine administration activates monoaminergic pathways and increases systemic corticosterone, both of which influence anxiety states and adult dentate gyrus neurogenesis. Chronic amphetamine increases anxiety states in rats when measured at 24 h and at 2 weeks of withdrawal. However, the effects of chronic amphetamine exposure and withdrawal on long term anxiety-like behavior ...
Known for Dentate Gyrus | Chronic Amphetamine | Anxiety States | Adult Neurogenesis
KOL-Index: 12374 . Withdrawal from amphetamine increases anxiety and reduces the ability to cope with stress, which are factors that are believed to contribute to drug relapse. Stress-induced serotonergic transmission in the central nucleus of the amygdala is associated with anxiety states and fear. Conversely, stress-induced increases in ventral hippocampal serotonin (5-HT) levels have been linked to coping ...
Known for Amphetamine Withdrawal | Ventral Hippocampus | Central Amygdala | 5ht Levels
KOL-Index: 12187 . Corticotropin-releasing factor (CRF) is a neurohormone that mediates stress, anxiety, and affects serotonergic activity. Studies have shown that CRF has dose-dependent opposing effects on serotonergic activity. This effect has been hypothesized to be differentially mediated by CRF(1) and CRF(2) receptors in the dorsal raphé nucleus. We directly tested this hypothesis by using in vivo ...
Known for Nucleus Accumbens | Serotonin Release | 2 Receptors | Releasing Factor
KOL-Index: 12115 . Withdrawal from amphetamine is associated with increased anxiety and sensitivity to stressors which are thought to contribute to relapse. Rats undergoing amphetamine withdrawal fail to exhibit stress-induced increases in serotonin (5-HT) release in the ventral hippocampus and show heightened anxiety-like behaviors. Therefore, we tested the hypothesis that reducing 5-HT levels in the ...
Known for Ventral Hippocampus | Amphetamine Withdrawal | Anxiety Behavior | 5 Ht
KOL-Index: 11684 . Central serotonergic neurons have been implicated in numerous animal behaviors and psychiatric disorders, but the molecular mechanisms underlying their development are not well understood. Here we generated Lmx1b (LIM homeobox transcription factor 1 beta) conditional knock-out mice (Lmx1b(f/f/p)) in which Lmx1b was only deleted in Pet1 (pheochromocytoma 12 ETS factor-1)-expressing 5-HT ...
Known for Central Serotonergic Neurons | Knockout Mice | Expression Lmx1b | Locomotor Activity
KOL-Index: 11322 . The effects of the serotonin neurotoxin 5,7-dihydroxytryptamine (5,7-DHT), on serotonin1 (5-HT1) and 5-HT2 receptors were investigated using the high degree of resolution provided by quantitative autoradiography in an effort to determine the synaptic location of these receptors. 5,7-DHT treatment resulted in a decrease in 5-HT1 binding in the dentate gyrus and CA3c/4 of the anterior ...
Known for Quantitative Autoradiography | Central Nervous | 5ht2 Receptors | 5 Ht1
KOL-Index: 10528 . Social stress in adolescence is correlated with emergence of psychopathologies during early adulthood. In this study, the authors investigated the impact of social defeat stress during mid-adolescence on adult male brain and behavior. Adolescent male Sprague-Dawley rats were exposed to repeated social defeat for 5 days while controls were placed in a novel empty cage. When exposed to ...
Known for Limbic Monoamines | Male Rats | Social Defeat | Anxiety Behavior
KOL-Index: 9930 . Microdissection techniques were utilized to measure the activity of choline acetyltransferase (ChAT) (enzyme responsible for synthesis of acetylcholine) in individual basal forebrain nuclei of aged (24 month) and young (4 month) male and female rats. Small but consistent decreases in the activity of ChAT in aged rats were found, and the location of the changes was dependent on the sex of ...
Known for Basal Forebrain | Activity Chat | Choline Acetyltransferase | Aged Rats
KOL-Index: 9843 . To assess whether the brain's monoaminergic and/or corticotropin-releasing factor (CRF) systems may be involved in mediating the appetite-suppressing effects of high environmental ammonia levels, we exposed rainbow trout to one of four NH4Cl treatments (0, 500, 750, 1000 micromol l(-1)) for 24 or 96 h and monitored changes in food intake, brain serotonin (5-HT) and dopamine (DA) activity, ...
Known for Ammonia Exposure | Rainbow Trout | Food Intake | Suppressing Effects
KOL-Index: 9462 . Stress induced by early life social isolation leads to long-lasting alterations in stress responses and serotonergic activity. Corticotropin-releasing factor (CRF) is a neurotransmitter that mediates stress responses and alters serotonergic activity. We tested the hypothesis that the stress of early life isolation enhances responses to CRF in adulthood by determining the effect of CRF ...
Known for Adult Rats | Social Isolation | Nucleus Accumbens | Early Life
KOL-Index: 9460 . Rapid activation of central serotonergic systems occurs in response to the social stress of aggression in dominant lizards. The most rapid expression of serotonergic activity occurs in nucleus accumbens, hippocampus and brainstem. To compare previously measured responses induced by social stressors with those provoked by physical stress, serotonergic activity was examined following ...
Known for Serotonergic Activity | Nucleus Accumbens | Physical Stress | Locus Ceruleus
KOL-Index: 9460 . In the rat dorsomedial hypothalamus (DMH), serotonin (5-HT) concentrations are altered rapidly in response to acute stressors. The mechanism for rapid changes in 5-HT concentrations in the DMH is not clear. We hypothesize that the mechanism involves corticosteroid-induced alterations in the uptake of 5-HT from extracellular fluid through the action of corticosterone-sensitive organic ...
Known for Medial Hypothalamus | Organic Cation | 5 Ht | Extracellular Fluid
KOL-Index: 9307 . The role of GABAergic neurons in activating female sexual behavior and possible mechanisms for GABAergic effects on behavior were examined in female rats. First, effects of the ovarian hormones estrogen and progesterone (P), at doses which promote lordosis, on levels and turnover/activity of GABA, were examined in brain areas which regulate lordosis. Utilizing AOAA, an inhibitor of GABA ...
Known for Female Rats | Gabaergic Neurons | Estrogen Progesterone | Gaba Vmn

Key People For Amphetamine Withdrawal

Top KOLs in the world
#1
George F Koob
nucleus accumbens animal models drug addiction
#2
Athina Markou
nicotine withdrawal brain reward function wistar receptors
#3
Larry Kokkinidis
acoustic startle locomotor activity electrical stimulation
#4
Manit Srisurapanont
alcohol dependence major depressive disorder amphetamine withdrawal
#5
Alasdair M Barr
antipsychotic drugs insulin resistance psychotic symptoms
#6
Robert J Barrett
drug discrimination training dose electroconvulsive shock

Department of Biology, University of South Dakota, Vermillion, SD 57069 USA | Neuroscience Group, Division of Basic Biomedical Sciences, Sanford School of Medicine, University of South Dakota, Vermillion, SD 57069 USA | Department of Biology, Univers