Nobuhiro Yuki

Nobuhiro Yuki

From The Department Of Neurology (Drs. Kuwabara, Asahina, Mori, And Hattori), Chiba University School Of Medicine, Chiba; And The Department Of Neurology (Drs. Koga And Yuki), ...

Direct Impact

Concepts for which Nobuhiro Yuki has direct influence:

guillain-barré syndrome
miller fisher
campylobacter jejuni
acute motor axonal neuropathy
axonal degeneration
igg antibody
conduction block

External impact

Concepts related to the work of other authors for which Nobuhiro Yuki has influence:

guillain-barré syndrome
southern china
campylobacter jejuni
conduction block
14 provinces
acute motor axonal neuropathy
axonal degeneration

Prominent publications by Nobuhiro Yuki

KOL-Index: 212 Several distinct autoimmune neuropathies can be referred to collectively as Guillain–Barré syndrome (GBS). Wakerley and colleagues argue that confusing terminology hinders diagnosis, and they present a revised classification of 'classic' GBS, Miller Fisher syndrome and related conditions. This new approach to diagnosis might facilitate work-up, thereby helping to identify the correct ...
Known for
Confusing Terminology | Fisher Syndromes | Classic Gbs | Revised Classification
KOL-Index: 75 BACKGROUND: Whether or not antiganglioside antibodies are related to axonal or demyelinating Guillain-Barré syndrome (GBS) is still a matter of controversy, as detailed in previous studies conducted in Western and Asian countries. OBJECTIVE: To clarify whether antiganglioside antibodies are associated with axonal dysfunction in Japanese and Italian GBS patient cohorts. METHODS: Clinical and ...
Known for
Axonal Dysfunction | Electrodiagnostic Criteria | N103 | Aman Electrodiagnosis
KOL-Index: 53 Guillain-Barré syndrome (GBS) is the commonest cause of acquired flaccid paralysis in the world and regarded by many as the prototype for postinfectious autoimmunity. Here the authors consider both infectious and noninfectious triggers of GBS and determine where possible what immunological mechanisms may account for this association. In approximately two-thirds of cases, an infectious ...
Known for
Syndrome Gbs | Triggers Guillain | Infectious Noninfectious | Reported Lead
KOL-Index: 53 BACKGROUND AND PURPOSE: This study aimed to investigate geographical differences in the clinical features of Guillain-Barré syndrome (GBS) between patients from our region in Eastern China and patients from other areas. METHODS: A total of 595 patients fulfilling the diagnostic criteria ​for GBS or its variants were included from two large hospitals located in Eastern China. Data collection ...
Known for
Interquartile Range Age | Antecedent Region | Pure Motor Predominant Gbs | Frequency Axonal
KOL-Index: 49 In the last few years, a lot of publications suggested that disabling cerebellar ataxias may develop through immune-mediated mechanisms. In this consensus paper, we discuss the clinical features of the main described immune-mediated cerebellar ataxias and address their presumed pathogenesis. Immune-mediated cerebellar ataxias include cerebellar ataxia associated with anti-GAD antibodies, the ...
Known for
Anti-Gad Antibodies | Cell-Mediated Immunity | Miller Fisher Syndrome | Humoral
KOL-Index: 43 Guillain-Barré syndrome is classified into acute inflammatory demyelinating polyneuropathy and acute motor axonal neuropathy. Whereas autoantibodies to GM1 or GD1a induce the development of acute motor axonal neuropathy, pathogenic autoantibodies have yet to be identified in acute inflammatory demyelinating polyneuropathy and chronic inflammatory demyelinating polyneuropathy. This review ...
Known for
Physiopathology | Polyneuropathy Chronic | Guillain-Barré Syndrome | Hereditary Demyelinating Neuropathies
KOL-Index: 35 The possibility that some patients diagnosed with an acute sensory neuropathy could actually have Guillain-Barré syndrome (GBS) has been repeatedly advanced in the literature, but the number of cases reported is small. The reports have shown different clinical presentations and electrophysiological findings and are variously named, thus generating terminological and nosological confusion. We ...
Known for
Sensory Fiber | Ganglionopathy | Syndrome Gbs | Neuropathy Guillain
KOL-Index: 34 The identification of autoantibodies associated with dysimmune neuropathies was a major contribution to the characterization of peripheral nerve disorders, the understanding of their pathophysiology, and the clinical diagnosis of neuropathies. Antibodies directed to GM1, GQ1b, and disyalilated gangliosides, and anti-MAG antibodies are very useful in the diagnosis of acute or chronic motor or ...
Known for
Antibody Peripheral | Motor Neuropathies | Disorders Understanding
KOL-Index: 34 This review charts the progress of anti-glycolipid antibodies in neuropathy, from their original discovery 20 years ago in immunoglobulin M paraproteinaemic neuropathy through to current discoveries mapping their relationship to subtypes of Guillain-Barré syndrome. Antibodies to >20 different glycolipids have now been associated with a wide range of clinically identifiable acute and chronic ...
KOL-Index: 34 Certain species of anti-ganglioside antibodies are associated with specific clinical features in various neurologic diseases. Serum autoantibodies to these minor gangliosides were investigated in a number of neurological diseases in order to examine the biological functions of GD1alpha and GQ1beta. Eleven patients with Guillain-Barré syndrome had remarkably high IgG anti-GD1alpha antibody ...
Known for
Igg Anti-Gd1alpha Antibody | Anti-Gq1b Activity | Target Molecules | Reacts

From the Department of Neurology (Drs. Kuwabara, Asahina, Mori, and Hattori), Chiba University School of Medicine, Chiba; and the Department of Neurology (Drs. Koga and Yuki), Dokkyo University School of Medicine, Tochigi, Japan.

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