KOL | Simon G TalbotDepartment of Radiation Oncology, Memorial Sloan-Kettering Cancer Center |
KOL Resume for Simon G Talbot
| Year | |
|---|---|
| 2008 | Department of Radiation Oncology, Memorial Sloan-Kettering Cancer Center |
Simon G Talbot: Influence Statistics
| Concept | World rank |
|---|---|
| human cne2 cells | #9 |
| human cne2 | #14 |
| dadsinduced apoptosis | #67 |
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Prominent publications by Simon G Talbot
In this study, we investigated whether the inhibition of endogenous phosphorylation of mitogen-activated protein kinase (MAPK) and diallyl disulfide (DADS)-induced phosphorylation of MAPKs with MAPK specific inhibitors, SB203580 and U0126 (for phospho-p38 and phospho-p42/p44, respectively), can induce or enhance apoptosis in human CNE2 nasopharyngeal carcinoma cells. Our data demonstrate that MAPK inhibitors decrease the viability of CNE2 cells, stimulate typical apoptotic morphologic ...
| Known for Cne2 Cells | Activated Protein | Induced Apoptosis | Kinase Mapk | Inhibitors Sb203580 |
Key People For Cne2 Cells
Simon G Talbot:Expert Impact
Concepts for whichSimon G Talbothas direct influence:Cne2 cells, Human cne2 cells, Human cne2, Dadsinduced apoptosis, Cne2 cell, Dads induced.
Simon G Talbot:KOL impact
Concepts related to the work of other authors for whichfor which Simon G Talbot has influence:Human cne2 cells, Korean children, Mycobacterium immunogenum, Secular trends, Alveolar macrophage, Chromone alkaloid, Hypersensitivity pneumonitis.
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