George T Gallagher: Influence Statistics

George T Gallagher

George T Gallagher

Division of Oral Pathology, Boston University Henry M. Goldman School of Dental Medicine, Boston, MA, USA | Division of Oral Pathology, Boston University School of Dental ...


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George T Gallagher: Expert Impact

Concepts for which George T Gallagher has direct influence: Messenger rna , Situ hybridization , Oral cancer , Salivary gland , Transforming growth , Tgf alpha , Growth factor .

George T Gallagher: KOL impact

Concepts related to the work of other authors for which for which George T Gallagher has influence: Oral cancer , Growth factor , Squamous cell , Wound healing , Situ hybridization , Polycystic adenosis , Head neck .

KOL Resume for George T Gallagher

Year
2016

Division of Oral Pathology, Boston University Henry M. Goldman School of Dental Medicine, Boston, MA, USA

2010

From the G. R. Harrison Spectroscopy Laboratory, Massachusetts Institute of Technology (McGee, Mirkovic, Yu, Badizadegan, Feld), the Departments of Otolaryngology—Head and Neck Surgery (Mardirossian, Elackattu, Wang, Grillone) and Anatomic Pathology (Pistey), Boston Medical Center, the Department of Oral and Maxillofacial Pathology, Boston University Goldman School of Dental Medicine (Gallagher, Kabani), and the Department of Pathology, Harvard Medical School and Massachusetts General Hospital (Badizadegan), Boston, Massachusetts.

2009

Boston Medical Center, the Department of Oral and Maxillofacial Pathology, Boston University Goldman School of Dental Medicine, Boston, Massachusetts.

2008

Boston University Goldman School of Dental Medicine, Department of Oral and Maxillofacial Pathology, 100 East Newton Street G-04, Boston, Massachusetts 02118

2007

Professor, Department of Oral and Maxillofacial Pathology, Boston University School of Dental Medicine, Boston, MA.

2006

Boston University School of Dental Medicine, USA.

2005

Boston University Goldman School of Dental Medicine, USA.

2002

Department of Oral Pathology, Boston University Goldman School of Dental Medicine, 100 E. Newton Street, Boston, MA 02118, USA

2001

Department of Oral Medicine and Diagnostic Sciences, Division of Oral Pathology, 188 Longwood Avenue, Harvard School of Dental Medicine, MA 02115, Boston, Massachusetts, USA

2000

HARVARD SCHOOL OF DENTAL MEDICINE AND BRIGHAM AND WOMEN’S HOSPITAL, aAssistant Professor, Dept of Oral Medicine and Diagnostic Sciences, Harvard School of Dental Medicine, Brigham and Women’s Hospital, bResearch Assistant, Department of Internal Medicine, Brigham and Women’s Hospital, cProfessor, Dept of Medicine, Harvard Medical School, Brigham and Women’s Hospital, dAssociate Professor, Department of Oral Medicine and Diagnostic Sciences, Harvard School of Dental Medicine

Boston, Mass

1999

Laboratory of Molecular Pathology, Division of Oral Pathology, Department of Oral Medicine and Diagnostic Sciences, Harvard School of Dental Medicine, Boston, MA 02115, USA

1998

Division of Oral Pathology, Department of Oral Medicine and Diagnostic Sciences, Harvard School of Dental Medicine, Boston, MAUSA

1997

Harvard School of Dental Medicine. Boston, Mass., USA

1996

Department of Oral Medicine and Diagnostic Sciences, Harvard School ofDental Medicine, Boston, Massachusetts 02215, USA.

1993

Department of Oral Pathology, Massachusetts General Hospital and Harvard School of Dental Medicine, 188 Longwood Avenue, 02115, Boston, MA, USA

1991

Department of Oral Medicine and Oral Pathology, Harvard School of Dental Medicine, 188 Longwood Avenue, Boston, Massachusetts 02115

1990

Department of Oral Medicine and Oral Pathology, Harvard School of Dental Medicine, Boston, Massachusetts 02115.

Prominent publications by George T Gallagher

KOL-Index: 10365 . Transforming growth factor-alpha (TGF-alpha) and TGF-beta 1 have been proposed as important regulators of processes critical to successful wound healing. Although various cells present in wounds represent potential sources of either TGF-alpha and/or TGF-beta, including macrophages, neutrophils, keratinocytes, fibroblasts, and endothelial cells, we recently identified eosinophils as an ...
Known for Wound Healing | Transforming Growth | Beta 1 | Tgf Alpha
KOL-Index: 9632 . Transforming growth factor alpha (TGF-alpha) is a pleuripotential cytokine with diverse biological effects, including the ability to influence the proliferation of normal cells or neoplastic epithelial cells. Eosinophils are a subset of granulocytes that normally enter the peripheral tissues, particularly those beneath gastrointestinal, respiratory, and urogenital epithelium, where they ...
Known for Human Eosinophils | Transforming Growth | Messenger Rna | Hypereosinophilic Syndrome
KOL-Index: 8626 . Using hamster as an oral wound healing model, we examined eosinophils and their expression of transforming growth factor-alpha (TGF-alpha) and transforming growth factor-beta 1 (TGF-beta 1). Oral wounds healed approximately two times faster than their cutaneous counterparts. Eosinophils infiltrated prominently into oral wounds; however, unlike the dual expression of TGF-alpha and TGF-beta ...
Known for Oral Wounds | Alpha Tgf | Growth Factor | Wound Healing
KOL-Index: 7839 . Aberrant expression of TGF-alpha is associated with human malignant oral epithelium. Experiments were initiated to determine the cellular sources of transforming growth factor-alpha (TGF-alpha) in human oral cancer. Ten freshly resected human oral cancers and four specimens of normal human oral epithelium were studied by in situ hybridization and immunohistochemistry. Tissues were probed ...
Known for Human Oral Cancer | Growth Factor | Tgf Alpha | Epithelial Cells
KOL-Index: 7365 . Carcinoma associated fibroblasts (CAFs) form the main constituents of tumor stroma and play an important role in tumor growth and invasion. The presence of CAFs is a strong predictor of poor prognosis of head and neck squamous cell carcinoma. Despite significant progress in determining the role of CAFs in tumor progression, the mechanisms contributing to their activation remain poorly ...
Known for Squamous Cell | Tumor Carcinoma | Caf Markers | Collagen Type
KOL-Index: 7075 . Inflammatory mediators of periradicular lesions are poorly understood. Transforming growth factors-alpha and -beta 1 (TGF-alpha and TGF-beta 1) have been linked with the cellular processes for both soft and hard tissue wound healing. The purpose of this study is to demonstrate the cellular sources of TGF-alpha and TGF-beta 1 mRNA and protein in periapical lesions by in situ hybridization ...
Known for Transforming Growth | Periapical Granulomas | Tgf Beta | Inflammatory Mediators
KOL-Index: 7053 . We recently demonstrated that eosinophils infiltrate prominently into cutaneous wounds in the Syrian hamster and represent a source of transforming growth factor-alpha and transforming growth factor-beta. In this study, we assessed the role of the eosinophil and eosinophil-derived transforming growth factors in human oral ulcers that exhibit delayed healing, descriptively termed traumatic ...
Known for Stromal Eosinophilia | Transforming Growth | Traumatic Ulcerative Granuloma | Human Eosinophils
KOL-Index: 6743 . Sclerosing polycystic adenosis (SPCA) of the salivary gland is a rarely encountered lesion of uncertain pathogenesis that shares histologic features with sclerosing adenosis and fibrocystic disease of the mammary gland. To date, fewer than 40 cases of SPCA have been reported in the literature; all but one have arisen in the major salivary glands. We report 3 cases of SPCA involving minor ...
Known for Salivary Gland | Polycystic Adenosis | Cysts Diagnosis | Differential Female
KOL-Index: 6470 . Large scale gene expression profiling was carried out on laser capture microdissected (LCM) tumor and normal oral epithelial cells and analysed on high-density oligonucleotide microarrays. About 600 genes were found to be oral cancer associated. These oral cancer associated genes include oncogenes, tumor suppressors, transcription factors, xenobiotic enzymes, metastatic proteins, ...
Known for Oral Cancer | Laser Capture | Gene Expression | Reverse Transcriptase
KOL-Index: 6258 . Epidermal growth factor (EGF) and transforming growth factor-alpha (TGF-alpha) are known to promote the healing of epithelial wounds. Eosinophils are present in healing wounds and have recently been shown to be capable of producing TGF-alpha. This investigation was done to determine if eosinophils infiltrated into healing wounds are capable of expressing this cytokine. Using the rabbit ...
Known for Growth Factor | Healing Wound | Cellular Source | Alpha Tgf
KOL-Index: 5427 . Wound healing is critical to the survival of the species after injury. Using hamsters as an experimental model, we have shown that eosinophils infiltrate prominently into skin wounds and that they express transforming growth factor-alpha and -beta 1 mRNAs and proteins. We hypothesized that eosinophils are important in wound healing. As no animal model is genetically deficient in ...
Known for Eosinophil Infiltration | Wound Healing | Monoclonal Antibody | Experimental Model

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Division of Oral Pathology, Boston University Henry M. Goldman School of Dental Medicine, Boston, MA, USA | Division of Oral Pathology, Boston University School of Dental Medicine, Boston, Massachusetts, United States of America. | From the G. R. Har