Emil J Bardana
Oregon Health and Science University, Portland, Ore | Oregon Health and Science University, Portland, OR 97239, USA. | Division of Allergy and Clinical Immunology, Department ...
KOL Resume for Emil J Bardana
Oregon Health and Science University, Portland, Ore
Oregon Health and Science University, Portland, OR 97239, USA.
Division of Allergy and Clinical Immunology, Department of Medicine, Oregon Health and Science University, 3181 SW Sam Jackson Park Road, OP34, Portland, OR 97239, USA
Oregon Health & Sciences University, Portland, Oregon.
Oregon Health and Sciences University, Division of Allergy and Clinical Immunology, 3181 Southwest, Sam Jackson Park Road, OP 34, Portland, OR 97201, USA.
Division of Allergy and Clinical Immunology, Oregon Health Sciences University, Portland, Oregon.
Division of Allergy and Clinical Immunology, Oregon Health Sciences University, Portland 97201-3098, USA.
Division of Allergy and Clinical Immunology, Oregon Health Sciences University, 97201-3098, Portland, OR
Department of Medicine, Oregon Health Sciences University, Portland.
Department of Medicine, Division of Allergy and Clinical Immunology The Oregon Health Sciences University, Portland, Oregon
Division of Allergy and Clinical Immunology, Oregon Health Sciences University, Portland 97201.
Professor of Medicine and Head, Division of Allergy and Clinical Immunology, Oregon Health Sciences University, Portland, Oregon
Oregon Health Sciences University, Division of Allergy and Clinical Immunology, Portland.
Oregon Health Sciences University, Portland, Oregon, USA
3181 S. W. Sam Jackson Park Road, 97201, Portland, OR
Oregon Health Sciences University, in Portland. He has evaluated numerous complaints of building sickness and is working to develop objective criteria for measuring its symptoms
Division of Immunology, Allergy and Rheumatology, Oregon Health Sciences University, Portland, Ore. USA
Oregon Health Sciences University, Portland, OR 97201, USA
Portland, Oregon, USA
From the Department of Medicine, Divisions of Nephrology and Immunology, and Department of Pathology, The Oregon Health Sciences University, Portland, Oreg.
University of Oregon Health Sciences Center Portland, Oregon, U.S.A.
and Health Sciences Center, University of Oregon, Portland, Oregon, United States
Oregon Regional Primate Research Center, Beaverton, Oregon 97006, U.S.A..
Division of Immunology, Allergy, and Rhematology Health Science Center, University of Oregon Portland, Oregon
From the Veterans Administration Hospital, U.S.A.
Univ. of Oregon Health Sciences Center, Portland, USA
Division of Immunology, Allergy and Rheumatology, University of Oregon Health Sciences Centre, Portland, Oregon, USA
Established Investigator of the American Heart Association La Jolla, California, USA
Divisions of Vascular Surgery and Immunology and the Department of Diagnostic Radiology, University of Oregon Health Sciences Center, Portland, Oregon
Department of Microbiology and Immunology and the Division of Immunology and Allergy, University of Oregon Health Sciences Center, Portland, Oreg.
From the Division of Immunology, Allergy, and Rheumatology, University of Oregon Health Sciences Center Portland, Ore., USA
Section of Allergy and Clinical Immunology, National Jewish Hospital and Research Center, Denver, Colorado, 80206 Division of Immunology and Allergy, University of Oregon Medical School, Portland, Oregon, 97201, USA
Division of Immunology and Allergy, University of Oregon Medical School, Portland, Oregon 97201
Division of Immunology and Allergy University of Oregon Medical School Portland, Oregon 97201
Portland, Oregon USA
From the National Jewish Hospital and Research Center Denver, Colo., USA
Departments of Allergy and Clinical Immunology and Clinical Laboratories, National Jewish Hospital and Research Center, Denver, Colorado 80206
Division of Immunology, Allergy and Infectious Diseases, University of Oregon Medical School, Portland, Ore.
Research Fellow of the Allergy Foundation of America.
Emil J Bardana: Influence Statistics
|positive cultures trial||#1|
|aspergillus normal subjects||#1|
|animal fish protein||#1|
|proposed1 aspergillus antibody2||#1|
|patients immunological responsiveness||#1|
|fungal antigens immunity||#1|
|ammonium sulfate test||#1|
|8 occupational asthma||#1|
|antinuclear antibody effects||#1|
|normal individuals binding||#1|
|mctd continental ancestry||#1|
|7 11 workers||#1|
|systemic lupuslike disease||#1|
|immunodiffusion fungal antigens||#1|
|primates primates amino||#1|
|aspergillin serum concentrations||#1|
|pathologic hallmark vasculitis||#1|
|abpa serum igd||#1|
|species radial immunodiffusion||#1|
|conidial antigens aspergillus||#1|
|neoplasia potential triad||#1|
|definite etiologic associations||#1|
|disease process battery||#1|
|pulmonary colonization infection||#1|
|candida antibody immunodiffusion||#1|
|triad natural evolution||#1|
|spectrum aspergillus antigens||#1|
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Prominent publications by Emil J Bardana
The primary interaction of antibody to components of aspergilli II. Antibodies in sera from normal persons and from patients with aspergillosis
[ PUBLICATION ]
The capacity of antibodies in 102 human sera to bind components of aspergilli was studied. Sera were from patients with pulmonary aspergillosis or a variety of other clinical disorders and from healthy individuals. The antigens employed were anionic mucopolysaccharide components derived from A. fumigatus and A. nidulans and were not precipitated by antibodies in either human or rabbit sera. The antigens were soluble in 10 per cent trichloroacetic add and 50 per cent saturated ammonium ...
|Known for Primary Interaction | Normal Persons | Antibodies Sera | Patients Aspergillosis | Antigens Fungal|
As summarized in Table 1, the literature consistently supports the hypothesis that allergic asthmatic patients have seasonal BHR changes that parallel allergen exposure. These seasonal changes appear to be preventable by treatment with corticosteroids (systemic, inhaled, or nasal), disodium cromoglycate, and immunotherapy. Studies have almost exclusively focused on pollens, though similar limited data exist for dust mites. Though the dust mite is a perennial allergen, mite levels are ...
|Known for Seasonal Variation | Perennial Rhinitis | Bronchial Hyperreactivity | Viral Infections | Dust Mite|
Antibody to DNA was measured before and after treatment of systemic lupus erythematosus (SLE) sera with bovine pancreatic deoxyribonuclease (DNase I). In 11 of 15 cases of SLE with active renal disease there was a significant increase in DNA-binding after DNase digestion, while no such increase was noted in inactive SLE, normal controls or in patients with nonlupus renal disease. The significant rise in DNA-binding after digestion indicated that DNA had bound in vivo to the anti-DNA in ...
|Known for Systemic Lupus | Dna Complexes | Erythematosus Sera | Renal Disease | Sle Nephritis|
Reactive airways dysfunction syndrome (RADS): guidelines for diagnosis and treatment and insight into likely prognosis
[ PUBLICATION ]
Reactive airways dysfunction syndrome (RADS) is defined as the sudden onset of asthma following a high level exposure to a corrosive gas, vapor, or fume. This variant of occupational asthma continues to generate controversy regarding the criteria for its diagnosis. There is also some disagreement as to the likely prognosis with this disorder. Currently, the diagnosis requires the assumption of normal premorbid pulmonary physiology and absence of bronchial hyperreactivity. Criteria for ...
|Known for Reactive Airways | Dysfunction Syndrome | Occupational Asthma | Bronchial Hyperreactivity | Sudden Onset|
Occupational asthma and related respiratory disorders
[ PUBLICATION ]
Occupational rhinitis is a common but generally underreported entity. Although it may occur alone, it is frequently associated with occupational asthma. Occupational asthma may have one of several presentations that are difficult to distinguish from non-work conditions. The respiratory tract acts as the final common pathway for all inhaled environmental pollutants, whether encountered in the home or at work. More than 200 chemicals have been incriminated as a cause of work-related ...
|Known for Occupational Asthma | Respiratory Tract | Rhinitis Common | Diagnosis Differential | Environmental Pollutants|
Effects of phenytoin on man's immunity Evaluation of changes in serum immunoglobulins, complement, and antinuclear antibody
[ PUBLICATION ]
To determine the effects of phenytoin on serum immunoglobulins, complement, and antinuclear antibody conversion, a prospective, five-year longitudinal study was undertaken in 118 patients. Three major diagnostic groups were evaluated: 27 patients with idiopathic epilepsy, 50 with secondary epilepsy, and 41 with neuropathic syndromes without epilepsy. In addition, 83 normal volunteers were studied in a similar manner. Evaluations were performed prior to administration of phenytoin and at ...
|Known for Serum Iga | Idiopathic Epilepsy | Effects Phenytoin | Antinuclear Antibodies | 27 Patients|
Antigens from Mycobacterium bovis and Listeria monocytogenes bind to serums from normal rabbits as well as from those immunized with unrelated bacteria, especially intracellular parasites. Binding is to the Fab portion of immunoglobulin G and is immunologically specific.
|Known for Mycobacterium Bovis | Shared Antigens | Listeria Monocytogenes | Bacterial Species | Unrelated Bacteria|
Antigenic Differences Between Mycobacterium bovis Strain BCG and an Isoniazid-Resistant Mutant.
[ PUBLICATION ]
The antigenicities of Mycobacterium bovis strain BCG and an isoniazid (INH) - resistant mutant were compared. Sonic extracts of heat-killed, washed bacteria were injected into rabbits, and antisera were produced (anti-BCG and anti-BCG-R). The sonic extracts were also subjected to ultracentrifugation, and test antigens were prepared from the resulting supernatants (BCG and BCG-R). The antigens were labeled with (131)I, and the antisera were tested for their capacity to bind (131)I-BCG and ...
|Known for Strain Bcg | Test Antigens | Mycobacterium Bovis | Inhibition Studies | Isoniazid Inh|
The primary interaction of antibody to components of aspergilli I. Immunologic and chemical characteristics of a nonprecipitating antigen
[ PUBLICATION ]
The experiments presented in this report describe the preparation of a radiolabeled antigen which can be used in the ammonium sulfate test to measure antibodies against aspergilli. The antigenic components were in the trichloroacetic acid (TCA)-soluble fraction from sonically disrupted aspergilli. The major radiolabeled ligand appeared to be an immunologically nonprecipitating anionic mucopolysaccharide which was insensitive to DNase or RNase, slightly sensitive to pepsin, and very ...
|Known for Primary Interaction | Bacterial Antigens | Genus Aspergillus | Antigen Antibody | Antibodies Fungal|
Aspergillus Antibody in Patients With Cystic Fibrosis
[ PUBLICATION ]
The respiratory flora of patients with cystic fibrosis (CF) frequently includes Aspergillus, and 30% of their serum samples have been observed to contain precipitating antibody to this fungus. Serum from 61 CF patients, 60 healthy persons, and three patients with CF and allergic bronchopulmonary aspergillosis was studied, using a quantitative assay for antibody to A fumigatus. Although CF patients had significantly higher levels of Aspergillus antibody, some antibody was found in all ...
|Known for Cystic Fibrosis | Fumigatus Patients | Immunoglobulin Infant | Aspergillus Antibody | Antibodies Fungal|
Key People For Occupational Asthma
Emil J Bardana:Expert Impact
Concepts for whichEmil J Bardanahas direct influence:Occupational asthma, Sick building syndrome, Digital ischemia, Myasthenia gravis, Serum igd, Autoimmune hemolytic anemia, Aspergillus antibody, Hand ischemia.
Emil J Bardana:KOL impact
Concepts related to the work of other authors for whichfor which Emil J Bardana has influence:Systemic lupus erythematosus, Aspergillus fumigatus, Immune complexes, Cystic fibrosis, Lupus nephritis, Occupational asthma, Sick building syndrome.
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